Insulin resistance and metabolism: organ crosstalk

The goal of this program is to implement an inventory of the different animal models, tissues and cell cultures used.

Model of crosstalk between diverse organs and pancreatic beta cells under both physiological and insulin-resistant states. The different factors represented may have the potential to enhance glucose-stimulated insulin secretion or beta-cell proliferation (as IGF-1—insulin-like growth factor-1; GLP-1—glucagon-like peptide-1; kisspeptin1 and SCFAs—short-chain fatty acids) or negatively contribute to reduce functional beta-cell mass during type 2 diabetes progression (as leptin, CXCL10 and EVs—extracellular vesicles—from inflamed adipocytes or insulin-resistant muscle cells). 

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Diabetes Institute scientific day

Diabetes Institute scientific day

The overall goal of this Diabetes Institute scientific day is to provide the most important and up-to-date research in the field of metabolism made at University Paris Cité. The workshop will focus on understanding the recent progress in adipose tissue and liver biology including metabolic and inflammatory processes in the control of the energy homeostasis. Special emphasis will be done to highlight the importance of the organ crosstalk and how signaling pathways in one tissue could affect the metabolism in other tissue.